Dr Ghada Elayat
| Name | Dr Ghada Elayat |
|---|---|
| Job title | Lecturer in Medical Sciences |
| Research institute | |
| Primary appointment | Natural Sciences |
| Email address | g.elayat@mdx.ac.uk |
| ORCID | https://orcid.org/0000-0002-8198-4309 |
| Contact category | Academic staff |
Biography
Biography Dr Ghada Elayat MBChB,
PhD, CSci, FIBMS, FHEA My journey in medicine began in Egypt, where I initially trained as a histopathologist. Later, I continued my training at the Histopathology Department, St Bartholomew's Hospital. While pursuing my PhD at the University of London, I also registered with the General Medical Council for a period to continue my training in Histopathology. After completing my PhD in 2006, I decided to shift my career towards academia and continued my career as a histopathologist and lecturer. In 2013, I joined MDX as a lecturer specialising in histopathology and clinical sciences. In 2022, I took on the role of Programme Leader for the BSc Medical Science programme, which has been running for two years now, with our first cohort set to graduate in 2025. My PhD studies were focused on breast cancer biology with special interest in apocrine metaplastic changes and their relation to cancer development. My work in this area has resulted in several publications in peer-reviewed journals. As my research evolved, I shifted my focus to bladder cancers, with a particular emphasis on the complexities of the tumour microenvironment. In addition, I have explored the domains of chronic inflammation and actively contributed to research involving the antimicrobial peptides pain, and periodontal status. My collective research work in the past six years has resulted in further publications in peer-reviewed journals, highlighting the continuous contributions to the advancement of knowledge in the field of cancer biology. As a Fellow of the Institute of Biomedical Science (FIBMS) and a Chartered Scientist, I am deeply committed to upholding the highest professional standards in my field. Serving as an IBMS mentor, I am dedicated to nurturing the growth and development of fellow scientists, fostering a culture of excellence in both research and practice.
Teaching Programme leader BSc Medical Science Module leader: BMS2125, BMS2515, BMS3326 I teach at both undergraduate/ postgraduate level and across several programmes. Modules taught at undergraduate level: BMS1014,
BMS2125, BMS2515, BMS3136, BMS3326 and BMS3336 and BIO3419.
Modules taught at postgraduate level: BMS4217,
BMS4237, BMS4507, BMS4547, BMS4227, BMS4117, BMS4137, BMS4997 and BMS4147 Supervisor of MSc, MRes and PhD projects.
Employment
Education and qualifications
Grants
Prizes and Awards
CPD Diploma
2024-07-10
IBMS
External activities
Research outputs
Phenotypic plasticity in melanoma: the impact on management strategies
Elayat, G. 2024. Phenotypic plasticity in melanoma: the impact on management strategies. Redactive Publishing.An overview of angiogenesis in bladder cancer
Elayat, G., Punev, I. and Selim, A. 2023. An overview of angiogenesis in bladder cancer. Current Oncology Reports. 25 (7), pp. 709-728. https://doi.org/10.1007/s11912-023-01421-5Positive correlational shift between crevicular antimicrobial peptide LL-37, pain and periodontal status following non-surgical periodontal therapy. A pilot study
Madruga, D., Garcia, M., Martino, L., Hassan, H., Elayat, G., Ghali, L. and Ceballos, L. 2023. Positive correlational shift between crevicular antimicrobial peptide LL-37, pain and periodontal status following non-surgical periodontal therapy. A pilot study. BMC Oral Health. 23 (1), p. 335. https://doi.org/10.1186/s12903-023-03023-wPhytochemical modulation of apoptosis and autophagy: strategies to overcome chemoresistance in leukaemic stem cells in the bone marrow microenvironment
Owen, H., Appiah, S., Hasan, N., Ghali, L., Elayat, G. and Bell, C. 2017. Phytochemical modulation of apoptosis and autophagy: strategies to overcome chemoresistance in leukaemic stem cells in the bone marrow microenvironment. in: Zeng, B. and Zhao, K. (ed.) Neurobiology of Chinese Herb Medicine Elsevier.Cyclin D-1 protein over-expression is not associated with gene amplification in benign and atypical apocrine lesions of the breast
Elayat, Ghada, Selim, Abdel-Ghani A., Gorman, Patricia, Tomlinson, Ian and Wells, Clive A. 2011. Cyclin D-1 protein over-expression is not associated with gene amplification in benign and atypical apocrine lesions of the breast. Pathology - Research and Practice. 207 (2), pp. 75-78. https://doi.org/10.1016/j.prp.2010.06.003Cell turnover in apocrine metaplasia and apocrine adenosis of the breast
Elayat, G., Selim, A. and Wells, C. 2010. Cell turnover in apocrine metaplasia and apocrine adenosis of the breast. Annals of Diagnostic Pathology. 14 (1), pp. 1-7. https://doi.org/10.1016/j.anndiagpath.2009.05.001Non-operative breast pathology: Apocrine lesions
Wells, C. and Elayat, G. 2007. Non-operative breast pathology: Apocrine lesions. Journal of Clinical Pathology. 60 (12), pp. 1313-1320. https://doi.org/10.1136/jcp.2006.040626Alterations of the cell cycle regulators cyclin D1, cyclin A, p27, p21, p16, and pRb in apocrine metaplasia of the breast
Elayat, G., Selim, A. and Wells, C. 2009. Alterations of the cell cycle regulators cyclin D1, cyclin A, p27, p21, p16, and pRb in apocrine metaplasia of the breast. Breast Journal. 15 (5), pp. 475-482. https://doi.org/10.1111/j.1524-4741.2009.00762.xCell cycle alterations and their relationship to proliferation in apocrine adenosis of the breast
Elayat, G., Selim, A. and Wells, C. 2009. Cell cycle alterations and their relationship to proliferation in apocrine adenosis of the breast. Histopathology. 54 (3), pp. 348-354. https://doi.org/10.1111/j.1365-2559.2009.03223.xC-myc oncoprotein expression and gene amplification in apocrine metaplasia and apocrine change within sclerosing adenosis of the breast
Selim, A., Elayat, G., Naase, M. and Wells, C. 2002. C-myc oncoprotein expression and gene amplification in apocrine metaplasia and apocrine change within sclerosing adenosis of the breast. The Breast. 11 (6), pp. 466-472. https://doi.org/10.1054/brst.2002.0474Expression of c-erbB2, p53, Bcl-2, Bax, c-myc and Ki-67 in apocrine metaplasia and apocrine change within sclerosing adenosis of the breast
Selim, A., Elayat, G. and Wells, C. 2002. Expression of c-erbB2, p53, Bcl-2, Bax, c-myc and Ki-67 in apocrine metaplasia and apocrine change within sclerosing adenosis of the breast. Virchows Archiv. 441, p. 449–455. https://doi.org/10.1007/s00428-002-0691-0Loss of heterozygosity and allelic imbalance in apocrine metaplasia of the breast: Microdissection microsatellite analysis
Selim, A., Ryan, A., Elayat, G. and Wells, C. 2002. Loss of heterozygosity and allelic imbalance in apocrine metaplasia of the breast: Microdissection microsatellite analysis. The Journal of Pathology. 196 (3), pp. 287-291. https://doi.org/10.1002/path.1043c-erbB2 oncoprotein expression, gene amplification, and chromosome 17 aneusomy in apocrine adenosis of the breast
Selim, A., Elayat, G. and Wells, C. 2000. c-erbB2 oncoprotein expression, gene amplification, and chromosome 17 aneusomy in apocrine adenosis of the breast. The Journal of Pathology. 191 (2), pp. 138-142. https://doi.org/10.1002/(sici)1096-9896(200006)191:2<138::aid-path611>3.0.co;2-jAngiogenesis in breast cancer: insights and innovations
Elayat, G. and Selim, A. 2024. Angiogenesis in breast cancer: insights and innovations. Clinical and Experimental Medicine. 24 (1). https://doi.org/10.1007/s10238-024-01446-5750
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